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Binge Eating Disorder is an eating disorder characterized by recurrent episodes of consuming an unusually large amount of food within a specific short period of time (typically within two hours or less), significantly more than most people would consume in the same time frame. During these episodes, individuals report a loss of control over eating and find it difficult to stop. A distinguishing feature of Binge Eating Disorder (BED) is the absence of compensatory behaviors such as vomiting, excessive exercise, or other purging actions following the binge episodes. In this regard, BED differs clinically from other eating disorders such as bulimia nervosa.
BED was first described by Albert Stunkard in 1959, and diagnostic criteria were later developed by Spitzer and colleagues in 1991. In the American Psychiatric Association’s Diagnostic and Statistical Manual of Mental Disorders (DSM), BED was initially included as a provisional diagnosis in DSM-IV and was formally recognized as a distinct diagnosis under the category “Feeding and Eating Disorders” in DSM-5, published in May 2013.
According to DSM-5 criteria, a diagnosis of Binge Eating Disorder requires the presence of recurrent binge eating episodes at least once a week for three months. During these episodes, the individual consumes an amount of food that is significantly larger than what most people would eat in a similar period and experiences a sense of lack of control over eating. Negative emotions such as extreme fullness, physical discomfort, guilt, shame, or distress often occur during or after the episodes.
BED is among the most common eating disorders in adults. The lifetime prevalence varies globally between 0.7% and 6.6%, with approximately 2.8% reported in the United States. It is more common in women than in men (about 1.75 times more frequent). The disorder typically begins in the twenties and may persist into middle age. Prevalence is higher among individuals with obesity, particularly among those receiving weight management treatment and those eligible for bariatric surgery.
The etiology of Binge Eating Disorder (BED) is understood to involve an interaction of genetic, biological, psychological, and environmental factors. Twin studies indicate a heritable component, with heritability estimates ranging from 41% to 74%. Neurotransmitter systems including dopamine, serotonin, opioids, and ghrelin have been implicated in BED. Additionally, functional abnormalities in the brain’s reward system are proposed to be associated with binge eating behavior.
Psychological factors include traumatic events experienced in early life and negative beliefs about the self. Clinical observations reveal that individuals diagnosed with BED frequently exhibit low self-esteem, negative body image, difficulties with emotional regulation, impulsivity, and behavioral patterns linked to addiction.
In Binge Eating Disorder (BED), individuals commonly experience a loss of control during eating episodes. They typically consume large quantities of food rapidly, often without feeling hungry. Following these episodes, individuals frequently report negative emotions such as guilt, shame, or regret, and it is commonly observed that they engage in these eating behaviors in private. BED is associated with weight gain and obesity, which can contribute to various physical health problems.
Psychiatric disorders commonly co-occurring with BED include major depressive disorder, anxiety disorders, and certain personality disorders (avoidant, borderline, obsessive-compulsive). Additionally, medical conditions related to obesity such as type 2 diabetes and cardiovascular disease are frequently present.
Longitudinal studies have shown that BED often follows a chronic course. While symptoms may diminish over time in some individuals, others may transition into other eating disorders.
The goals of BED treatment include cessation of binge eating episodes, establishment of regular eating patterns, achievement of weight stability, and management of co-occurring psychiatric conditions.
Warning: The content in this article is provided solely for general encyclopedic informational purposes. The information presented here should not be used for diagnosis, treatment, or medical advice. Always consult a physician or qualified healthcare professional before making any decisions regarding health. The author and KÜRE Encyclopedia assume no responsibility for any consequences arising from the use of this information for diagnostic or therapeutic purposes.
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Diagnosis and History
Epidemiology
Etiology
Clinical Features
Course and Prognosis
Treatment